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美国Seracare β2糖蛋白 IGM（Beta 2 Glycoprotein IgM）
广州健仑生物科技有限公司
广州健仑长期供应各种生物原料，主要代理品牌：美国Seracare、西班牙Certest、美国Fuller等等。
主要产品包括各种标准品、阳性对照品、单克隆抗原抗体。
其中常见的有：弓形虫病、西尼罗河病毒、类风湿因子、疟疾、麻疹、莱姆病、百日咳杆菌、大肠杆菌、鼠伤寒沙门氏菌、李斯特菌等阳性对照品。
美国Seracare β2糖蛋白 IGM（Beta 2 Glycoprotein IgM）
我司还提供其它进口或国产试剂盒：登革热、疟疾、流感、A链球菌、合胞病毒、腮病毒、乙脑、寨卡、黄热病、基孔肯雅热、克锥虫病、违禁品滥用、肺炎球菌、军团菌、化妆品检测、食品安全检测等试剂盒以及日本生研细菌分型诊断血清、德国SiFin诊断血清、丹麦SSI诊断血清等产品。
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【Seracare产品介绍】
编号 | 英文名称 | 中文名称 |
JL-FA-01 | Amebiasis (AME) | 阿米巴病 |
JL-FA-02 | Allergens, Rast scores | 过敏原，放射性过敏原吸收实验。指对特定的人群引起免疫反应或者过敏反应的食品中的蛋白质 |
JL-FA-03 | Allergens, Rast scores negative | 过敏原，放射性过敏原吸收实验阴性 |
JL-FA-04 | Anti-cyclic citrullinated peptide Antibody (CCP) Arthritis | 抗环瓜氨酸肽抗体 |
JL-FA-05 | ASCA Saccharomyces Cerevi | 人抗酿酒酵母抗体（ASCA） |
JL-FA-06 | Aspergillis | 麴菌病 |
JL-FA-07 | Beta 2 Glycoprotein | β2糖蛋白 |
JL-FA-08 | Beta 2 Glycoprotein IgM | β2糖蛋白 IGM |
JL-FA-09 | Bordela Pertussis | 百日咳杆菌 |
JL-FA-10 | Bordela Pertussis IgM | 百日咳杆菌 IGM |
JL-FA-11 | C-ANCA | C-抗中性粒细胞胞浆抗体（ANCA) |
JL-FA-12 | Cardiolipin | 心肌磷脂 |
JL-FA-13 | Cardiolipin IgA | 心肌磷脂 IGA |
JL-FA-14 | Cardiolipin IgG | 心肌磷脂 IGG |
JL-FA-15 | Cardiolipin IgM | 心肌磷脂 IGM |
JL-FA-16 | Cerebral Spinal Fluid | 脑脊髓液 |
JL-FA-17 | Chagas | 恰加斯病/南美锥虫 |
JL-FA-18 | Chlamydia | 衣原体 |
JL-FA-19 | Chlamydia IgA | 衣原体IGA |
JL-FA-20 | Chlamydia IgG | 衣原体IGG |
JL-FA-21 | Chlamydia IgM | 衣原体IGM |
JL-FA-22 | Chlamydia Neg | 衣原体阴性 |
JL-FA-23 | Clotting Factor C3 | 凝固因子C3 |
JL-FA-24 | Clotting Factor C4 | 凝固因子C4 |
JL-FA-25 | Coccidiodes | 球孢菌 |
JL-FA-26 | Cytomegalovirus (CMV) Neg | 巨细胞病毒抗体阴性 |
JL-FA-27 | CMV IgG | 巨细胞病毒 IGG阳性 |
JL-FA-28 | CMV IgM VCA | 巨细胞病毒 IGM 阳性 |
JL-FA-29 | C-Reactive Protein (CRP) | C-反应蛋白质 |
JL-FA-30 | Dengue Fever | 登革热 |
JL-FA-31 | Dengue Fever IgM | 登革热 IGM |
JL-FA-32 | DS (Double Stranded) DNA | 双链脱氧核糖核酸 |
JL-FA-33 | EBNA (Epstein-Barr nuclear antigen) IgG | EB病毒核抗原 IGG |
JL-FA-34 | EBNA (Epstein-Barr nuclear antigen) IgM | EB病毒核抗原 IGM |
JL-FA-35 | Epstein Barr Virus (EBV) Negative Plasma | EB病毒阴性血浆 |
JL-FA-36 | Epstein Barr Virus (EBV) EA IgM | EB病毒早期抗原 IGM |
JL-FA-37 | Epstein Barr Virus (EBV) VCA IgM | EB病毒壳蛋白 IGM |
JL-FA-38 | Epstein Barr Virus (EBV) EA IgG | EB病毒早期抗原 IGG |
JL-FA-39 | EMA (Endomysial Antibodies) | 肌内膜 |
JL-FA-40 | Gliadin | 麸蛋白,麦醇溶蛋白,麦胶蛋白 |
JL-FA-41 | Gliadin IgG | 麦醇溶蛋白 IGG |
JL-FA-42 | Gliadin IgA | 麦醇溶蛋白 IGA |
JL-FA-43 | Glomerular Basement Membrane (GBMA) | 肾小球基底膜病 |
JL-FA-44 | Helicobacter pylori IgA | 幽门螺旋杆菌IGA |
JL-FA-45 | Helicobacter pylori IgG | 幽门螺旋杆菌IGG |
JL-FA-46 | Helicobacter pylori IgM | 幽门螺旋杆菌IGM |
JL-FA-47 | Helicobacter pylori Negative | 幽门螺旋杆菌阴性 |
JL-FA-48 | Helicobacter pylori Positive Plasma | 幽门螺旋杆菌阴性血浆 |
JL-FA-49 | Hepatitis A Virus (HAV) Pos. Plasma | 甲型肝炎病毒阳性血浆 |
JL-FA-50 | Hepatitis A Virus (HAV) IgM | 甲型肝炎病毒IGM |
JL-FA-51 | Hepatitis B Core (HBc) IgG | 乙型肝炎病毒核心 IGG |
JL-FA-52 | Hepatitis B Core (HBc) IgM | 乙型肝炎病毒核心 IGM |
JL-FA-53 | Anti Hbe (Antibody to HBV antigen) | 乙肝抗体 |
JL-FA-54 | Hepatitis Delta Virus | 丁型肝炎病毒 |
JL-FA-55 | HBeAg (HBV e antigen) | 乙肝 E抗原 |
JL-FA-56 | anti-HBs (HBV surface antibody) | 乙肝表面抗体 |
JL-FA-57 | Hepatitis B (HBsAg) "Chronic" | 乙型肝炎（乙肝表面抗原）“慢性病 |
JL-FA-58 | HBsAg (HBV surface antigen) Serum | 乙肝表面抗原血清 |
JL-FA-59 | HBsAg (AD) | 乙肝表面抗原（AD） |
JL-FA-60 | HBsAg (AY) | 乙肝表面抗原（AY） |
JL-FA-61 | HBV Positive Plasma | 乙肝阳性血浆 |
JL-FA-62 | HBV DNA Plasma | 乙肝DNA血浆 |
JL-FA-63 | HBV DNA Serum | 乙肝DNA血清 |
JL-FA-64 | HBV DNA type A | A型 乙肝DNA |
JL-FA-65 | HBV DNA type B | B型 乙肝DNA |
JL-FA-66 | HBV DNA type C | C型 乙肝DNA |
JL-FA-67 | HBV DNA type D | D型 乙肝DNA |
JL-FA-68 | HBV DNA type E | E型 乙肝DNA |
JL-FA-69 | HBV DNA type F | F型 乙肝DNA |
JL-FA-70 | HBV Antibody HCV Antibody Plasma CO-INFECTED | 乙肝和丙肝联合感染血浆 |
JL-FA-71 | HCV (Hepatitis C Virus) Antibody | 丙型肝炎抗体 |
JL-FA-72 | HCV Core Antigen Positive | 丙肝核心抗原 阳性 |
JL-FA-73 | HCV RNA PLASMA Genotype 1 | 基因1型丙肝RNA 血浆 |
JL-FA-74 | HCV RNA PLASMA Genotype 2 | 基因2型丙肝RNA 血浆 |
JL-FA-75 | HCV RNA PLASMA Genotype 3 | 基因3型丙肝RNA 血浆 |
JL-FA-76 | HCV RNA PLASMA Genotype 4 | 基因4型丙肝RNA 血浆 |
JL-FA-77 | HCV RNA PLASMA Genotype 5 | 基因5型丙肝RNA 血浆 |
JL-FA-78 | HCV RNA PLASMA Genotype 6 | 基因6型丙肝RNA 血浆 |
JL-FA-79 | HCV Riba single band | 丙肝免疫印迹单波段 |
JL-FA-80 | HCV RIBA Pos. (multiple bands) | 丙肝免疫印迹阳性多波段 |
JL-FA-81 | HCV Negative | 丙肝阴性 |
JL-FA-82 | HCV RNA Pos (quantitative) | 丙肝RNA阳性（定量） |
JL-FA-83 | Hepatitis E | 戊型肝炎 |
JL-FA-84 | Herpes Simplex Virus (HSV)1/2 Positive Plasma | 单纯性疱疹病毒1/2阳性血浆 |
JL-FA-85 | Herpes Simplex Virus (HSV) 1 Negative Plasma | 单纯性疱疹病毒1 阴性血浆 |
JL-FA-86 | Herpes Simplex Virus (HSV) 1 IgG | 单纯性疱疹病毒1 IGG |
JL-FA-87 | Herpes Simplex Virus (HSV 1) IgM | 单纯性疱疹病毒1 IGM |
JL-FA-88 | Herpes Simplex Virus (HSV) 2 IgG | 单纯性疱疹病毒2 IGG |
JL-FA-89 | Herpes Simplex Virus (HSV) 2 IgM | 单纯性疱疹病毒2 IGG |
JL-FA-90 | Histone | 组蛋白 |
JL-FA-91 | Human Anti Mouse Ab (HAMA) | 人抗鼠抗体 |
JL-FA-92 | Human immunodeficiency virus (HIV) 1 Neg | HIV I 阴性 |
JL-FA-93 | anti Human immunodeficiency virus (HIV) 1 Plasma | 抗HIV I 血浆 |
JL-FA-94 | anti Human immunodeficiency virus (HIV) 1 Serum | 抗HIV I 血清 |
JL-FA-95 | anti Human immunodeficiency virus (HIV) 2 Western Blot Tested | 抗HIV 2 免疫印迹 |
JL-FA-96 | anti Human immunodeficiency virus (HIV) 1/2 2 HIV (+) | 抗HIV 1/2 2 HIV阳性 |
JL-FA-97 | Human immunodeficiency virus (HIV) Ag | HIV抗原 |
JL-FA-98 | HIV RNA (quantitative) Plasma | HIV RNA 定量血浆 |
JL-FA-99 | HIV RNA (quantitative) Serum | HIV RNA 定量血清 |
JL-FA-100 | HIV1 Subtype A | HIV1 亚型A |
JL-FA-101 | HIV1 Subtype B | HIV1 亚型B |
JL-FA-102 | HIV1 Subtype C | HIV1 亚型C |
JL-FA-103 | HIV1 Subtype D | HIV1 亚型D |
JL-FA-104 | HIV1 Subtype E | HIV1 亚型E |
JL-FA-105 | HIV1 Subtype F | HIV1 亚型F |
JL-FA-106 | HIV1 Subtype G | HIV1 亚型G |
JL-FA-107 | HIV1 Subtype H | HIV1 亚型H |
JL-FA-108 | HIV1 Subtype J | HIV1 亚型J |
JL-FA-109 | HIV1 Subtype K | HIV1 亚型K |
JL-FA-110 | HIV1 Group O | HIV1 亚型O |
JL-FA-111 | Human immunodeficiency virus (HIV) 2 Antibody Plasma | HIV 2 抗体血浆 |
JL-FA-112 | Human immunodeficiency virus (HIV) 2 Antibody Serum | HIV 2 抗体血清 |
JL-FA-113 | HPV (Human Papiloma Virus) Negative | 人乳状瘤病毒HPV阴性 |
JL-FA-114 | HPV (Human Papiloma Virus) Positive | 人乳状瘤病毒HPV阳性 |
JL-FA-115 | Human immunodeficiency virus (HIV) Antibody HCV Antibody Plasma COINFECTED | HIV 抗体 HCV |
JL-FA-116 | Human T-cell Lymphotropic Virus (HTLV) I/II | 人嗜T淋巴细胞病毒(HTLV) I/II |
JL-FA-117 | Human T-cell Lymphotropic Virus (HTLV) I | 人嗜T淋巴细胞病毒(HTLV) I |
JL-FA-118 | Human T-cell Lymphotropic Virus (HTLV) II | 人嗜T淋巴细胞病毒(HTLV) II |
JL-FA-119 | Jo-1 | 多发性肌炎抗原JO-1 |
JL-FA-120 | IgE < 5,000 Ku/L | IgE < 5,000 Ku/L |
JL-FA-121 | Legionella | 军团杆菌属 |
JL-FA-122 | Leptospira | 军团杆菌属 |
JL-FA-123 | Lyme Disease | 莱姆(氏)病:蜱传播的全身性疾病,常在夏季发生 |
JL-FA-124 | Lyme IgG | 莱姆(氏)病 IGG |
JL-FA-125 | Lyme IgM | 莱姆(氏)病 IGM |
JL-FA-126 | Lyme Disease Neg | 莱姆(氏)病 阴性 |
JL-FA-127 | Malaria | 疟疾 |
JL-FA-128 | Mononucleosis (infectious) | 单核细胞增多症（有传染性的） |
JL-FA-129 | Mononucleosis Negative | 单核细胞增多症阴性 |
JL-FA-130 | Measles Negative | 麻疹 阴性 |
JL-FA-131 | Measles IgG | 麻疹 IGG |
JL-FA-132 | Measles IgM | 麻疹 IGM |
JL-FA-133 | Microsomal Anti-thyroid peroxidase antibody (TPO) Positive Plasma Standard Titer (typically 1,000-3,000 IU/mL) | 微粒体抗甲状腺过氧化物酶抗体 |
JL-FA-134 | Microsomal Anti-thyroid peroxidase antibody (TPO) Negative Plasma | 微粒体抗甲状腺过氧化物酶抗体 |
JL-FA-135 | Anti-mitochondrial antibody (AMA) | 抗线粒体抗体 |
JL-FA-136 | Multiple Sclerosis | 多发性硬化症 |
JL-FA-137 | Mumps IgG | 流行性腮腺炎 IGG |
JL-FA-138 | Mumps Ab IgM | 流行性腮腺炎抗体 IGM |
JL-FA-139 | Mumps Antibody Negative Plasma | 流行性腮腺炎抗体阴性血浆 |
JL-FA-140 | Mumps Antibody Negative Serum | 流行性腮腺炎抗体阴性血清 |
JL-FA-141 | Myeloma Plasma | 骨髓瘤血浆 |
JL-FA-142 | Myeloma IgA | 骨髓瘤IGA |
JL-FA-143 | Myeloma IgE | 骨髓瘤IGE |
JL-FA-144 | Myeloma IgG | 骨髓瘤IGG |
JL-FA-145 | Myeloma IgM | 骨髓瘤IGM |
JL-FA-146 | Mycoplasma | 支原体 |
JL-FA-147 | Mycoplasma Negative | 支原体阴性 |
JL-FA-148 | Mycoplasma IgG | 支原体IGG |
JL-FA-149 | Mycoplasma IgM | 支原体IGM |
JL-FA-150 | Mycoplasma PCR | 支原体PCR |
JL-FA-151 | Normal Human Plasma | 正常人血浆 |
JL-FA-152 | Normal Human Serum | 正常人血清 |
JL-FA-153 | Nuclear Antibody Centromere | 核抗体着丝粒 |
JL-FA-154 | Nuclear Antibody, Speckled ANA | 核抗体，斑点抗核抗体 |
JL-FA-155 | Nuclear Antibody, Nucleolar ANA | 核抗体,核仁抗核抗体 |
JL-FA-156 | Nuclear Antibody, Homogeneous ANA | 核抗体，同质抗核抗体 |
JL-FA-157 | Nuclear Antiobody, Speckled. (ANA) Negative | 核抗体，斑点。抗核抗体阴性 |
JL-FA-158 | P-ANCA (associated neutrophil cytoplasmic antibodies) | 相关的嗜中性粒细胞胞浆抗体 |
JL-FA-159 | Parietal Cell Antibody (PCA) | 胃)壁细胞抗体 |
JL-FA-160 | Parvo positive plasma | 细小病毒阳性血浆 |
JL-FA-161 | Parvo IgM | 细小病毒 IGM |
JL-FA-162 | Parvo IgG | 细小病毒 IGG |
JL-FA-163 | Parvo Negative Plasma | 细小病毒阴性血浆 |
JL-FA-164 | Parvo DNA positive | 细小病毒 DNA 阳性 |
JL-FA-165 | Phospholipid Positive Plasma | 磷脂阳性血浆 |
JL-FA-166 | Prothrombin | 凝血酶原,凝血因子 |
JL-FA-167 | Rheumatoid Factor (RF) <1000 IU/mL | 类风湿因子<1000 IU/mL |
JL-FA-168 | Rheumatoid Factor (RF) 1001-2000 IU/mL | 类风湿因子1001-2000 IU/mL |
JL-FA-169 | Rheumatoid Factor (RF) 2001-4000 IU/mL | 类风湿因子 2001-4000 IU/mL |
JL-FA-170 | Rheumatoid Factor (RF) 4001-5000 IU/mL | 类风湿因子 4001-5000 IU/mL |
JL-FA-171 | Rheumatoid Factor (RF) >5000 IU/mL | 类风湿因子>5000 IU/mL |
JL-FA-172 | Ribonucleoprotein (RNP) Positive | 核糖核蛋白阳性 |
JL-FA-173 | Rubella Chimeric | 风疹 |
JL-FA-174 | Rubella Negative | 风疹阴性 |
JL-FA-175 | Rubella IgG | 风疹IGG |
JL-FA-176 | Rubella IgM | 风疹IGM |
JL-FA-177 | Rubeola Negative Plasma | 风疹阴性血浆 |
JL-FA-178 | Rubeola IgG | 风疹IGG |
JL-FA-179 | Scleroderma (Scl-70) Pos | 胶原沉着病,硬皮病,硬皮症 阳性 |
JL-FA-180 | Scleroderma (Scl-70) Negative | 硬皮病阴性 |
JL-FA-181 | Sickle Cell Fresh Whole Blood | 镰刀形红细胞新鲜全血 |
JL-FA-182 | Smith (SM) | 抗Smith抗体阳性血清（SLE的特征性抗体） |
JL-FA-183 | SMITH RNP | 抗RNP抗体阳性血清（SLE的特征性抗体） |
JL-FA-184 | Smooth Muscle (ASMA) | 抗平滑肌抗体阳性血清 |
JL-FA-185 | Sjogren syndrome antigen A (SSA) Positive | 舍格伦综合征或干燥综合征抗原A 阳性 |
JL-FA-186 | Sjogren syndrome antigen B (SSB) Positive | 舍格伦综合征抗原B 阳性 |
JL-FA-187 | Sjogren syndrome antigen B (SSB) Negative | 舍格伦综合征抗原B阴性 |
JL-FA-188 | Streptolysin O Ab (ASO) | 链球菌溶血素O抗体 |
JL-FA-189 | Syphilis (RPR - Rapid Plasma Reagin) Positive Plasma | 梅毒（梅毒-快速血浆反应）阳性血浆 |
JL-FA-190 | Syphilis (RPR - Rapid Plasma Reagin) Negative Plasma | 梅毒（梅毒-快速血浆反应）阴性血浆 |
JL-FA-191 | Syphilis/ATA/T. pallidum IgG | 梅毒ATA/T，苍白球IGG |
JL-FA-192 | Syphilis/ATA/T. pallidum IgM | 梅毒ATA/T，苍白球IGM |
JL-FA-193 | Systemic Lupus Erythematosus (SLE) Positive | 全身性红斑狼疮阳性 |
JL-FA-194 | Systemic Lupus Erythematosus (SLE) Negative | 全身性红斑狼疮阴性 |
JL-FA-195 | TG/TPO Positive (Standard Titer 1,000 - 3000 IU/mL) | 甲状腺球蛋白/甲状腺过氧化物酶阳性 |
JL-FA-196 | TG/TPO Negative | 甲状腺球蛋白/甲状腺过氧化物酶阴性 |
JL-FA-197 | TTG (Tissue Transglutaminase) | 组织转谷氨酰胺酶 |
JL-FA-198 | TTG (Tissue Transglutaminase) IgA | 组织转谷氨酰胺酶 IGA |
JL-FA-199 | ToRCH (Toxo, Rubella, CMV, HSV) Positive | 优生优育（弓形虫，风疹，巨细胞，单胞）阳性 |
JL-FA-200 | ToRCH (Toxo, Rubella, CMV, HSV) Negative | 优生优育（弓形虫，风疹，巨细胞，单胞）阴性 |
JL-FA-201 | Toxoplasmosis (Toxo) | 弓形虫病 |
JL-FA-202 | Toxoplasmosis (Toxo) IgG | 弓形虫病IGG |
JL-FA-203 | Toxoplasmosis (Toxo) IgM | 弓形虫病IGM |
JL-FA-204 | Thyroglobulin (TG) Positive Plasma | 甲状腺球蛋白阳性血浆 |
JL-FA-205 | Thyroglobulin (TG) Negative | 甲状腺球蛋白阴性 |
JL-FA-206 | Varicella-Zoster Virus (VZV) Negative | 水痘-带状疱疹病毒阴性 |
JL-FA-207 | Varicella-Zoster Virus (VZV) IgG | 水痘-带状疱疹病毒IGG |
JL-FA-208 | Varicella-Zoster Virus (VZV) IgM | 水痘-带状疱疹病毒IGM |
JL-FA-209 | West Nile Virus (WNV) | 西尼罗河脑炎病毒 |
JL-FA-210 | West Nile Virus (WNV) IgM | 西尼罗河脑炎病毒IGM |
美国Seracare β2糖蛋白 IGM（Beta 2 Glycoprotein IgM）
在这项研究中，怀特海德研究所的McKinley发现了两种确保CENP-A正确填充的关键激酶，Plk1和CDK。这两种激酶参与了CENP-A填充的不同步骤，只有它们都正常起作用，CENP-A才能填满着丝粒中的所有空隙。McKinley不仅解析了这些激酶的作用途径，还在此基础上干扰了CENP-A的填充时机，研究显示这种干扰会引起严重的染色体分离问题。
“着丝粒的功能处于严格的控制之下，因此人们一直认为CENP-A的填充时机应该很重要。现在，我们终于证实了这一理论，”McKinley说。
“CENP-A填充是着丝粒形成的核心步骤，”Cheeseman说，他也是MIT的生物学副教授。 “这项研究揭示了这一步骤的调控基础，有助于我们深入理解细胞分裂的具体过程。”
干细胞可替代中枢神经系统损伤后丢失的细胞，减少神经组织损害，促进功能恢复。许多脑损伤模型，如大脑中动脉阻塞和创伤性脑损伤模型中均证实神经干细胞可从脑室下区迁移至大脑皮质损伤区。但目前仍不够清晰的问题是，激活缺血大脑内源性神经干细胞的机制何在?
韩国全南国立大学医学院法医学系Hyung-Seok Kim博士所在课题组的研究揭示，局灶性脑缺血后神经干细胞的激活存在时序性，并验证了早期表达的低氧诱导因子1α和血管内皮生长因子组成的微环境提高了脑缺血后激活的内源性神经干细胞神经可塑性。大脑皮质损伤后，神经前体细胞的损失可由损伤周围区域和脑室下区得以补充。
大脑细胞的自我更新能力很差，细胞疗法可使丢失脑细胞的得以更新，已成为中枢神经系统损伤治疗的潜力性方法。骨髓间充质干细胞因其可分化为神经元/神经细胞，又可通过血脑屏障迁移至损伤神经组织，还能分泌神经营养因子，营造利于神经再生的微环境，也被认为是有希望的细胞治疗项目。
伊朗Shahid Sadoughi 大学医学院的Mohammad Ali Khalili教授所在研究团队，设计了给创伤性脑损伤模型大鼠尾静脉注射3×106大鼠骨髓间充质干细胞，静脉移植后显著促进了创伤性损伤大脑皮质神经细胞的再生，作者认为此方法可成为因损伤而丢失神经细胞的有益补充。相关研究成果发表在《中国神经再生研究(英文版)》杂志2014年5月第9期。
在人类细胞中，位于制造能量的称为线粒体的结构中的DNA 可能出现突变从而形成多个DNA变种。这种称为异质性的情况是一批疾病的原因，但是健康人群的线粒体基因组中的致病异质性的流行程度尚不清楚，这部分是由于样本数量少或者此前研究的不充分的测序方法。
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In this study, McKinley at the Whitehead Institute identified two key kinases, Plk1 and CDK, that ensure correct filling of CENP-A. These two kinases are involved in different steps of CENP-A packing, and only if they both function normally, CENP-A can fill all the voids in the centromere. Not only did McKinley interpret the pathway of action of these kinases, but they also interfered with the timing of filling CENP-A, and studies showed that this interference can cause serious chromosomal segregation problems.
"The centromere function is under tight control, so it has been argued that the timing of filling with CENP-A should be important, and we have finally confirmed that," McKinley said.
"CENP-A packing is a central step in centromere formation," said Cheeseman, who is also an associate professor of biology at MIT. "This study reveals the basis for the regulation and control of this step, helping us to understand the specific process of cell division."
Stem cells can replace lost cells after CNS injury, reduce nerve tissue damage and promote functional recovery. Many brain injury models, such as middle cerebral artery occlusion and traumatic brain injury models, have confirmed that neural stem cells can migrate from the subventricular zone to the cerebral cortical lesion. However, the question remains unclear: what is the mechanism of activation of endogenous neural stem cells in ischemic brain?
A study by Dr. Hyung-Seok Kim, MD, from the Department of Forensic Medicine, Jeonnam National University School of Medicine, South Korea, revealed that the activation of neural stem cells after focal cerebral ischemia is sequential and validates the early expression of hypoxia inducible factor-1α and vascular endothelial Microenvironment of growth factor enhances the neuroplasticity of endogenous neural stem cells activated after cerebral ischemia. After cortical injury, the loss of neural progenitor cells can be compensated for by the area surrounding the lesion and the subventricular zone.
Brain cells are poor at self-renewal and cell therapies that allow for the loss of brain cells have been renewed and have become a potential method of treatment for CNS injury. Bone marrow-derived mesenchymal stem cells are also considered promising because of their ability to differentiate into neurons / neurons, migration to damaged nerve tissue through the blood-brain barrier, secretion of neurotrophic factors, and creation of a microenvironment conducive to nerve regeneration Cell Therapy Project.
Professor Mohammad Ali Khalili, a professor at the Shahid Sadoughi University School of Medicine in Iran, designed a rat model of traumatic brain injury to inject 3 × 10 6 rat bone marrow mesenchymal stem cells into the tail vein of the traumatic brain injury model and significantly promote traumatic injury to the cerebral cortex Regeneration of nerve cells, the authors believe that this method can become a beneficial supplement due to injury and loss of nerve cells. Relevant research results published in the "Chinese Journal of Nervous Regeneration Research (English Edition)" magazine in May 2014 No. 9.
In human cells, DNA located in the structure called mitochondria that make energy can mutate to form multiple DNA variants. This condition, known as heterogeneity, is responsible for a number of diseases, but the prevalence of pathogenic heterogeneity in the mitochondrial genome of healthy populations is unclear, partly due to the small sample size or inadequay studied Sequencing method.
