美国Seracare鲑肾杆菌阳性对照

广州健仑生物科技有限公司

广州健仑长期供应各种生物原料，主要代理品牌：美国Seracare、西班牙Certest、美国Fuller等等。

主要产品包括各种标准品、阳性对照品、阳性质控品、单克隆抗原抗体。

其中常见的有：弓形虫病、西尼罗河病毒、类风湿因子、疟疾、麻疹、莱姆病、百日咳杆菌、大肠杆菌、鼠伤寒沙门氏菌、李斯特菌等阳性对照品。

美国Seracare鲑肾杆菌阳性对照

我司还提供其它进口或国产试剂盒：登革热、疟疾、流感、A链球菌、合胞病毒、腮病毒、乙脑、寨卡、黄热病、基孔肯雅热、克锥虫病、违禁品滥用、肺炎球菌、军团菌、化妆品检测、食品安全检测等试剂盒以及日本生研细菌分型诊断血清、德国SiFin诊断血清、丹麦SSI诊断血清等产品。

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【Seracare产品介绍】

美国Seracare

这项工作由美国国家癌症研究所(K99CA181352)和美国癌症协会资助。
当前的理论认为，情节记忆的编码是通过海马回路的早期重塑推动的，而大脑皮层的重塑发生几个星期到几个月以后，才促进长期记忆存储和回忆。
在本研究中，美国麻省理工学院等处的研究人员利用全基因组RNA测序，超微结构成像和全细胞记录在野生型小鼠平行研究表明，经历了恐惧条件作用的小鼠改变了基因转录，突触活动增加，并且大脑的中前额皮层(mPFC)区域内的可塑性增加，而抑制中前额皮层(mPFC)神经元能削弱长期记忆的形成，这提示了中前额皮层(mPFC)内的早期变化可能让长期记忆储存成为可能。
这些结果表明，长期情节记忆的编码与新皮层回路早期重塑有关，确定前额叶皮层作为编码引起的海马活化和长期记忆形成的关键调节器，以及对了解健康和患病的大脑状态的记忆过程具有重要意义。
在肿瘤组织中一种名为捎带细胞(Piggy-backing Cells)的快速生长的细胞往往会同高度侵袭性的细胞一起，一旦其到达机体的不同部位就会开启高效的侵袭模式，zui终形成新的肿瘤组织。文章中研究者利用透明斑马鱼进行研究，以便其可以观察到癌细胞如何在机体中移动以及如何进行侵袭。肿瘤细胞转移的恶果是造成癌细胞全身性扩散，侵略全身各处的组织器官。因此，想办法锁定控制这种转移或者是从一开始就阻断它们的信息传递将会是抗癌路上的一个突破性进展。
比利时鲁汶天主教大学的研究人员*成功的利用小鼠研究人类肿瘤模型，研究结果发表在了7月24日的 Cell Report 期刊上。
鲁汶天主教大学实验和临床研究中心的 Pierre Sonveaux's 教授带领及其所带领的团队前期对肿瘤细胞的线粒体进行了深入研究，进而成功发现了一个化学药物家族，发挥药效时能够防止肿瘤细胞的转移。他们发现线粒体这一 “细胞动力工厂”的功能改变时，会促进肿瘤细胞的转移。
博士后研究的 Paolo E. Porporato 和 Pierre Sonveaux's 团队中其他一些年轻的研究人员深入挖掘了线粒体这一功能的分子机制，zui终发现是由于线粒体在某些特定情况下，会产生大量的自由基——超氧化物，正是这些超氧化物的过剩导致肿瘤细胞的转移，造成不可挽回的后果。
包括帕金森病和阿尔茨海默氏症等许多其它人类疾病中，线粒体产生的超氧化物都可以被特殊的抗氧化剂所封闭，例如 MitoTEMPO 。小鼠给药以及人类肿瘤模型给药的结果均阐明了，这些化合物能够高效地阻断肿瘤细胞的迁移，并且防止小鼠体内人类肿瘤的自发形成。

美国Seracare

我司还提供其它进口或国产试剂盒：登革热、疟疾、流感、A链球菌、合胞病毒、腮病毒、乙脑、寨卡、黄热病、基孔肯雅热、克锥虫病、违禁品滥用、肺炎球菌、军团菌、食品安全、化妆品检测、药物滥用检测等试剂盒以及日本生研细菌分型诊断血清、德国SiFin诊断血清、丹麦SSI诊断血清等产品。

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【公司名称】 广州健仑生物科技有限公司
【市场部】    杨永汉

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【腾讯  】 2042552662
【公司地址】 广州清华科技园创新基地番禺石楼镇创启路63号二期2幢101-103室

This work is funded by the National Cancer Institute (K99CA181352) and the American Cancer Society.
Current theory suggests that the encoding of episodic memory is motivated by early remodeling of the hippocampal circuit, and that the remodeling of the cerebral cortex promotes long-term memory storage and recall after only a few weeks to several months.
In this study, researchers at the Massachusetts Institute of Technology and other institutes used genome-wide RNA sequencing, ultrastructural imaging and whole-cell recording in wild-type mice to study in parallel that mice experiencing the fear condition changed gene transcription , An increase in synaptic activity, and an increase in plasticity in the brain's region of the mid-prefrontal cortex (mPFC), while inhibition of mPFC neurons impairs long-term memory formation, suggesting an early phase within the mPFC Changes may make long-term memory storage possible.
These results indicate that long-term episodic memory coding correlates with early remodeling of neocortical circuits, determining the prefrontal cortex as a key regulator of coding-induced hippocampal activation and long-term memory formation, as well as memory processes for understanding healthy and diseased brain states It is of great significance.
In tumor tissue, a rapidly growing cell called Piggy-backing Cells, often with highly aggressive cells, turns on an efficient invasion pattern once it reaches different parts of the body, eventually forming a new tumor organization. In the article, researchers used transparent zebrafish to study how cancer cells can be observed in the body and how they can be infiltrated. Tumor cell metastasis is the result of the spread of cancer cells systemic invasion of tissues and organs throughout the body. Therefore, finding ways to lock in such transfers or blocking their messaging from the outset will be a breakthrough on the road to cancer.
Researchers at the Catholic University of Leuven in Belgium for the first time successfully used mice to study human tumor models, the findings of which were published in the July 24 issue of the Cell Report.
Led by Pierre Sonveaux's professor of the Center for Experimental and Clinical Studies at the Catholic University of Leuven and his team conducted an in-depth study on the mitochondria of tumor cells in advance and successfully discovered a family of chemical drugs that can prevent tumor cell metastasis . They found that when the function of mitochondria, a "cell-powered factory," changed, they promoted tumor cell metastasis.
Paolo E. Porporato, a postdoctoral researcher, and other young researchers at Pierre Sonveaux's team delve into the molecular mechanisms of mitochondrial function, culminating in mitochondria that produce large amounts of free radicals in certain cases Oxides, it is the excess of these superoxides that cause the metastasis of tumor cells, resulting in irreparable consequences.
In many other human diseases, including Parkinson's disease and Alzheimer's disease, the mitochondrial superoxide can be blocked by special antioxidants such as MitoTEMPO. The results of administration to mice as well as administration to human tumor models have demonstrated that these compounds are highly effective at blocking tumor cell migration and prevent the spontaneous formation of human tumors in mice.