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美国Seracare热灭活大肠杆菌O121:H19物种阳性对照
广州健仑生物科技有限公司
广州健仑长期供应各种生物原料,主要代理品牌:美国Seracare、西班牙Certest、美国Fuller等等。
主要产品包括各种标准品、阳性对照品、阳性质控品、单克隆抗原抗体。
其中常见的有:弓形虫病、西尼罗河病毒、类风湿因子、疟疾、麻疹、莱姆病、百日咳杆菌、大肠杆菌、鼠伤寒沙门氏菌、李斯特菌等阳性对照品。
美国Seracare热灭活大肠杆菌O121:H19阳性对照
我司还提供其它进口或国产试剂盒:登革热、疟疾、流感、A链球菌、合胞病毒、腮病毒、乙脑、寨卡、黄热病、基孔肯雅热、克锥虫病、违禁品滥用、肺炎球菌、军团菌、化妆品检测、食品安全检测等试剂盒以及日本生研细菌分型诊断血清、德国SiFin诊断血清、丹麦SSI诊断血清等产品。
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【Seracare产品介绍】
货号 | 中文名称 | 英文名称 |
JL-SC001 | 鼠伤寒沙门氏菌阳性对照 | Salmonella typhimurium Positive Control |
JL-SC002 | 志贺氏菌属阳性对照 | Shigella Species Positive Control |
JL-SC003 | 弧菌属阳性对照 | Vibrio Species Positive Control |
JL-SC004 | 军团菌嗜肺军团菌阳性对照 | Legionella pneumophila Positive Control |
JL-SC005 | BacTrace®金黄色葡萄球菌阳性对照 | BacTrace® Staphylococcus aureus Positive Control |
JL-SC006 | Bactrace®化脓性链球菌阳性对照 | BacTrace® Streptococcus pyogenes Positive Control |
JL-SC007 | bactrace®无乳链球菌阳性对照 | BacTrace® Streptococcus agalactiae Positive Control |
JL-SC008 | 李斯特菌属特异性阳性对照 | Listeria, Genus-Specific Positive Control |
JL-SC009 | 弯曲菌属特异性阳性对照 | Campylobacter, Genus-Specific Positive Control |
JL-SC010 | 幽门螺旋杆菌阳性对照 | Helicobacter pylori Positive Control |
JL-SC011 | 大肠杆菌O157:H7阳性对照 | Escherichia coli O157:H7 Positive Control |
JL-SC012 | BacTrace®大肠杆菌O111:H8物种阳性对照 | BacTrace® Escherichia coli O111:H8 Species Positive Control |
JL-SC013 | BacTrace®大肠杆菌O26:H11物种阳性对照 | BacTrace® Escherichia coli O26:H11 Species Positive Control |
JL-SC014 | Bactrace®大肠杆菌O103:H8的阳性对照,热灭活 | BacTrace® E.coli O103:H8 Positive Control, Heat-Killed |
JL-SC015 | Bactrace®大肠杆菌O145:H2的阳性对照,热灭活 | BacTrace® E.coli O145:H2 Positive Control, Heat-Killed |
JL-SC016 | Bactrace®大肠杆菌O121:H19的阳性对照,热灭活 | BacTrace® E.coli O121:H19 Positive Control, Heat-Killed |
JL-SC017 | Bactrace®大肠杆菌O45:H2的阳性对照,热灭活 | BacTrace® E.coli O45:H2 Positive Control, Heat-Killed |
JL-SC018 | BacTrace®大肠杆菌O104:H12阳性对照 | BacTrace® Escherichia coli O104:H12 Positive Control |
JL-SC019 | BacTrace®大肠杆菌O91阳性对照 | BacTrace® Escherichia coli O91 Positive Control |
JL-SC020 | 鲑肾杆菌阳性对照 | Renibacterium salmoninarum Positive Control |
美国Seracare热灭活大肠杆菌O121:H19阳性对照
当你期待的东西——比如你在餐厅点餐——或者引发你兴趣的东西,这时*的电节律就会席卷你的大脑。这些电波被称为伽马振荡,它们反映了细胞的交响曲,即兴奋性和抑制性相互协调。尽管γ波的作用一直有争论,但其已与更高级别的脑功能相关联,以及它的干扰模式已与精神分裂症,阿尔茨海默氏病,自闭症,癫痫症和其它疾病有关。
现在,索尔克研究所的新研究表明,小有名气的被称为星形胶质细胞的大脑支持细胞,实际上可能是控制这些电波的主要参与者。相关文章发表于2014年7月28日的《PNAS》杂志上。
索尔克研究人员报告了新的,意想不到的策略(通过禁用星形胶质细胞而不是神经元)减小伽马振荡。在这个过程中,研究组表明,星形胶质细胞有助于伽马振荡形成,这对于某些形式的记忆是至关重要的。
“这可以被称为一个确凿的证据,”合作者Terrence Sejnowski说。他是索尔克生物科学研究所计算神经生物学实验室的负责人和霍华德休斯医学研究所的研究员。“有数百篇论文认为伽马振荡和记忆力以及注意力有关。这是*,我们已经能够做一个因果的实验。通过选择性地阻断伽马振荡,研究表明,它对大脑与世界如何相互作用有一个非常具体的影响。”
索尔克研究所的Sejnowski教授,Inder Verma和斯蒂芬·海涅曼实验室合作, 在小鼠的大脑中,星形胶质细胞钙信号的活动形式紧跟在伽马振荡之前。这表明,星形胶质细胞,和神经元一样,使用许多相同的化学信号影响这些振荡。
为了验证他们的理论,该研究组使用携带破伤风毒素的病毒禁止星形胶质细胞选择性释放化学物质的能力,有效地消除了细胞与邻近细胞的沟通能力。神经细胞不受毒素的影响。
添加化学物质引发动物大脑的伽马波后,研究人员发现,携带有缺陷的星形胶质细胞的脑组织比含有健康细胞的组织产生更短的伽马波。而且,添加三个允许研究人员有选择地打开和关闭星形胶质细胞的破伤风毒素的基因后,他们发现,星形胶质细胞的信号传导被阻断的小鼠的伽玛波被削弱了。关闭毒素则抗原抗体了这一效果。
星形胶质细胞经过修改后的小鼠看似*健康的。但经过几个认知测试后,研究人员发现,它们缺乏一个主要领域:新物体识别。正如预期的那样,健康小鼠比熟悉的物品花更多的时间在置放于其环境中的新物品。与此相反,该研究组的新突变小鼠处理所有的物品的时间都是相同的。
“在某种意义上说,这原来是个惊人的结果,新物体识别记忆不只是受损,而是已经没了,就好像我们删除这一种形式的记忆,而其它完好无损,” Sejnowski说。
美国Seracare
我司还提供其它进口或国产试剂盒:登革热、疟疾、流感、A链球菌、合胞病毒、腮病毒、乙脑、寨卡、黄热病、基孔肯雅热、克锥虫病、违禁品滥用、肺炎球菌、军团菌、食品安全、化妆品检测、药物滥用检测等试剂盒以及日本生研细菌分型诊断血清、德国SiFin诊断血清、丹麦SSI诊断血清等产品。
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【公司名称】 广州健仑生物科技有限公司
【市场部】 杨永汉
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【腾讯 】 2042552662
【公司地址】 广州清华科技园创新基地番禺石楼镇创启路63号二期2幢101-103室
When you look for something - such as when you order at a restaurant - or something that interests you, the unique rhythm rolls over your brain. These waves are called gamma oscillations, and they reflect the symphony of the cell, the excitatory and inhibitory coordination of each other. Although the role of gamma-wave has been debated, it has been associated with higher levels of brain function and its mode of interference has been associated with schizophrenia, Alzheimer's disease, autism, epilepsy and other diseases .
Now, new studies at the Salk Institute show that the little-known brain-supporting cells, called astrocytes, could actually be the main players in controlling these waves. The article was published in the July 28, 2014 PNAS magazine.
Sork researchers reported new and unexpected strategies to reduce gamma oscillations by disabling astrocytes instead of neurons. In the process, the team showed that astrocytes help form gamma oscillations, which are crucial for some forms of memory.
"This can be called a conclusive evidence," said collaborator Terrence Sejnowski. He is the head of the computational neurobiology lab at the Salk Institute for Biological Sciences and a researcher at the Howard Hughes Medical Institute. "Hundreds of papers have argued that gamma oscillations are associated with memory and attention, and for the first time, we have been able to do a causal experiment, and by selectively blocking gamma oscillations, studies show how it interacts with the world The role has a very specific impact. "
In collaboration with Professor Sejnowski at the Salk Institute, Inder Verma, and Stephen Heinemann Labs, the astroglial calcium signaling activity in mice brains immediay follows gamma oscillations. This suggests that astrocytes, like neurons, affect many of these oscillations using many of the same chemical signals.
To validate their theory, the team used tetanus toxin-containing viruses to block the ability of astrocytes to selectively release chemicals, effectively eliminating the cells' ability to communicate with neighboring cells. Nerve cells are not affected by toxins.
After chemicals added to trigger the brain's gamma waves, the researchers found that brain tissue carrying defective astrocytes produced shorter gamma waves than tissues containing healthy cells. Moreover, after adding three genes that allow researchers to selectively open and close astrocytes in the tetanus toxins, they found that the gamma waves in mice blocked by astrocyte signaling were impaired It's Anti-toxin turned off this effect.
The modified astrocytes seem compley healthy. But after a few cognitive tests, the researchers found that they lacked one of the main areas: new object recognition. As expected, healthy mice spend more time placing new items in their environment than familiar items. In contrast, the new mutant mice in the study group treated all the items for the same amount of time.
"It turned out to be a surprising result in a sense that the new object recognition memory is not just damaged, but gone, just as if we deleted this form of memory while the rest were intact," Sejnowski said.