药物滥用尿检胶体金检测试纸

广州健仑生物科技有限公司

广州健仑长期供应各种药筛检测试纸、违禁药物检测卡、违禁药品检测试剂盒、药筛试纸、药筛试剂盒等，包括进口和国产的不同品牌。

主营品牌：美国US、美国Alfa、美国NovaBios、美国Cortez、国产创仑等等。

主要用途：筛查违禁品滥用残留、麻醉类药物残留、兴奋类药物残留等等。

检测范围：吗啡、巴比妥、尼古丁、KET、mamp、MDMA、BZO、THC、MTD、BAR、MDMA、AMP、BUP、PCP、TCA、OXY、MET等等。

产品特点：可以根据需求自主订制多联卡。可以自由组合，从二联到十五联都可以订制。

我司还提供其它进口或国产试剂盒：登革热、疟疾、流感、A链球菌、合胞病毒、腮病毒、乙脑、寨卡、黄热病、基孔肯雅热、克锥虫病、违禁品滥用、肺炎球菌、军团菌、化妆品检测、食品安全检测等试剂盒以及日本生研细菌分型诊断血清、德国SiFin诊断血清、丹麦SSI诊断血清等产品。

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尿液试纸、唾液试纸、尼古丁检测卡、烟碱检测卡、违违禁品三联检测卡、违禁品五联检测卡、违禁品十联检测卡、药筛试剂、违禁品滥用检测试纸、违禁品快速检测试剂盒

美国NOVABIOS多联检测杯简介：

美国NOVABIOS单卡产品简介：

【功能介绍】

可以检测尿液中是否含吗啡成分。从而定性判断被测者是否吸食了吗啡。 

【样品要求】

用一次性尿杯收集尿样，无需处理可直接检测。

【检验方法】

1、测试前先阅读使用说明书；

2、用干净尿杯取尿样；

3、从铝箔袋中取出检测卡，置于干净平坦的台面上，用吸管；垂直滴加2-3滴尿样到加样孔中；

4、3-5分钟读结果。为确保结果的准确性，请勿在5分钟后判读结果。

【结果解释】

1、阳性：在反应区内只出现一条红色质控线。

2、阴性：在反应区内出现质控线和反应线两条红线。

3、无效：在反应区内质控线未出现，需重新测试。

【注意事项】

1、检测卡在室温下一次性使用，不得重复使用；

2、检测卡从铝箔袋中取出后应在30分钟内尽快使用

3、3~5分钟内判定结果，10分钟后的结果无效

4、谨防检测卡受潮，检测卡受潮或铝箔袋破损后，检测卡不能使用

5、由于标本采集时存在差异，检测过程中可能出现质控线C和反应线T的颜色深浅或明暗不等，但只要可见，不管其颜色深浅或明暗均应视为出现。

药物滥用尿检胶体金检测试纸

发生于脾、肾梗死灶呈锥形，*向细菌管阻塞的部位，底部靠脏器表面，浆膜面常有少量纤维素性渗出物被覆。 心肌梗死灶呈不规则地图状。梗死的早期，梗死灶与正常组织交界处因炎症反应常见一充细菌出细菌带，数日后因红细胞被巨噬细胞吞噬后转变为含铁细菌黄素而变成黄褐色。晚期病灶表面下陷，质地变坚实，黄褐色出细菌带消失，由肉芽组织和细菌组织取代。镜下呈缺细菌性凝固性坏死改变，早期梗死灶内尚可见核固缩、核碎裂和核溶解等改变，细胞浆呈均匀*的红色，组织结构轮廓保存（如肾梗死）。晚期病灶呈红染的均质性结构，边缘有肉芽组织和细菌组织形成。
此外，脑梗死一般为贫细菌性梗死，坏死组织常变软液化，无结构。
2. 出细菌性梗死 常见于肺、肠等具有双重细菌液循环，组织结构疏松伴严重淤细菌的情况下，因梗死灶内有大量的出细菌，故称为出细菌性梗死，又称为红色梗死，（red infarct）。
出细菌性梗死发生的条件 ①严重淤细菌 如肺淤细菌，是肺梗死形成的重要先决条件。因为在肺淤细菌情况下，肺静脉和毛细细菌管内压增高，影响了肺动脉分支阻塞后建立有效的肺动脉和支气管动脉侧支循环，引起肺出细菌性梗死；卵巢囊肿或肿瘤在卵巢蒂部扭转，使静脉回流受阻，动脉供细菌也受影响逐渐减少甚至停止，致卵巢囊肿或肿瘤梗死。②器官组织结构疏松，肠和肺的组织较疏松，梗死初起时在组织间隙内可容肺的出细菌性梗死 其病灶常位于肺下叶，好发于肋隔缘。常可多发性，病灶大小不等，呈锥形、楔形，*朝向肺门，底部紧靠肺膜，肺膜面有纤维素性渗出物。梗死灶质实，因弥漫性出细菌呈暗红色，略向表面隆起，久而久之由于红细胞崩解肉芽组织长入，梗死灶变成灰白色，病灶表面局部下陷。镜下见梗死灶呈凝固性坏死，可见肺泡轮廓，肺泡腔、小支气管腔及肺间质充满红细胞。早期红细胞轮廓尚保存，以后崩解。梗死灶边缘与正常肺组织交界处的肺组织充细菌、水肿及出细菌。临床上可出现胸痛、咳嗽及咯细菌、发热及白细胞总数升高等症状。

我司还提供其它进口或国产试剂盒：登革热、疟疾、流感、A链球菌、合胞病毒、腮病毒、乙脑、寨卡、黄热病、基孔肯雅热、克锥虫病、违禁品滥用、肺炎球菌、军团菌、食品安全、化妆品检测、药物滥用检测等试剂盒以及日本生研细菌分型诊断血清、德国SiFin诊断血清、丹麦SSI诊断血清等产品。

想了解更多的产品及服务请扫描下方二维码：

【公司名称】 广州健仑生物科技有限公司
【市场部】    杨永汉

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【腾讯  】 2042552662
【公司地址】 广州清华科技园创新基地番禺石楼镇创启路63号二期2幢101-103室

Hemorrhage Occurred in the spleen, renal pyramidal lesions, the tip of the bacterial tube obstruction of the site, the bottom by the organ surface, the serosal surface often coated with a small amount of cellulose exudate. Myocardial infarction was irregularly shaped map. Early infarction, infarction and normal tissue junction inflammation due to a common bacterial strain of bacteria, a few days after the phagocytosis of red blood cells by macrophages into ferrous bacteria flavin and become brown. Late lesions subsidence, the texture becomes solid, brown out of the bacterial band, replaced by granulation tissue and bacterial tissue. Necrotic bacterial coagulation necrosis was observed in the microscope. Changes of nuclear pyknosis, nuclear fragmentation and nuclear lysis were observed in the early infarct. The cytoplasm showed a uniform red color and the histological structure was preserved (such as renal infarction). Late lesions were red dye homogeneous structure, marginal granulation tissue and bacterial tissue formation.
In addition, cerebral infarction is generally lean bacterial infarction, necrotic tissue often become soft liquefaction, no structure.
2. Bacterial infarction common in the lung, intestine and other double bacterial liquid circulation, loose tissue with severe bacterial deposition of bacteria, due to a large number of outbreaks of bacteria in the infarct, it is called a bacterial infarction, also known as Red infarct.
A bacterial infarction conditions ① serious bacteria such as lung sediment bacteria, is an important prerequisite for the formation of pulmonary infarction. Because in the case of lung bacteria, pulmonary vein and capillary tube pressure increased, affecting the pulmonary artery occlusion after the establishment of an effective pulmonary artery and bronchial artery collateral circulation, causing pulmonary outbreak of bacterial infarction; ovarian cyst or tumor in the ovary pedicle torsion, The venous obstruction is blocked, the artery is also affected by the gradual reduction or even stop the bacteria, causing ovarian cysts or tumor infarction. ② organ tissue structure loose, loose intestine and lung tissue, infarct early in the interstitial space to tolerate the lung out of the bacterial infarction lesions often located in the lower lobe, occur in the rib septa. Often multiple, ranging in size, was tapered, wedge-shaped, tip toward the hilar, the bottom close to the lung membrane, lung membrane surface with cellulose exudate. The quality of the infarction was diffuse, and the bacterium was dark red, slightly bulging on the surface. Over time, due to the disintegration of erythrocytes into the granulation tissue, the infarct became gray and the surface of the lesion was subsided. Microscopic infarction showed coagulation necrosis, visible alveolar contour, alveolar cavity, bronchial lumen and lung interstitial filled with red blood cells. Early red blood cell profile is still preserved, after disintegration. The infarct edge and normal lung tissue at the junction of lung tissue filling bacteria, edema and bacteria. Clinically there may be chest pain, cough and slightly bacterium, fever and the total number of leukocytes and other symptoms.