药物滥用快速检测试纸(胶体金法)
药物滥用快速检测试纸(胶体金法)

药物滥用快速检测试纸(胶体金法)

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广州健仑生物科技有限公司

广州健仑生物科技有限公司

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产品简介

药物滥用快速检测试纸(胶体金法) A rapid, one step test for the qualitative detection of Amphetamines in human urine. For healthcare professionals including professionals at point of care sites For professional i

详细介绍

药物滥用快速检测试纸(胶体金法)

广州健仑生物科技有限公司

广州健仑长期供应各种药筛检测试纸、违禁药物检测卡、违禁药品检测试剂盒、药筛试纸、药筛试剂盒等,包括进口和国产的不同品牌。

主营品牌:美国US美国Alfa、美国NovaBios、美国Cortez、国产创仑等等。

主要用途:筛查违禁品滥用残留、麻醉类药物残留、兴奋类药物残留等等。

检测范围:吗啡、巴比妥、尼古丁、KET、mamp、MDMA、BZO、THC、MTD、BAR、MDMA、AMP、BUP、PCP、TCA、OXY、MET等等。

产品特点:可以根据需求自主订制多联卡。可以自由组合,从二联到十五联都可以订制。

我司还提供其它进口或国产试剂盒:登革热、疟疾、流感、A链球菌、合胞病毒、腮病毒、乙脑、寨卡、黄热病、基孔肯雅热、克锥虫病、违禁品滥用、肺炎球菌、军团菌化妆品检测、食品安全检测等试剂盒以及日本生研细菌分型诊断血清、德国SiFin诊断血清、丹麦SSI诊断血清等产品。

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尿液试纸、唾液试纸、尼古丁检测、烟碱检测、违违禁品联检测、违禁品联检测、违禁品联检测药筛试剂、违禁品滥用检测试纸、违禁品快速检测试剂盒

美国NOVABIOS多联检测杯简介:

产品名称

规格

检测违禁品类型

违禁品十联检测杯

25T/盒

MET.AMP.MTD.THC.BAR.TCA.COC.BZO.PCP.OPI

违禁品十联检测杯

25T/盒

AMP.BAR.BZO.COC.MET.MOR.MTD.PCP.PPX.TCA.THC.XTC.WADU

违禁品十二联检测杯

25T/盒

BZO.BAR.COC.THC.MET.OPI.OXY.MDMA.PCP.AMP.BUP.MTD

 

美国NOVABIOS单卡产品简介:

品名称

英文缩写

检测阀值

吗啡 检测试剂盒

MOP(OPI)

300ng/ml

mamp 检测试剂盒

MAMP(MET)

1000ng/ml

K 检测试剂盒

KET

1000ng/ml

Ecstasy 检测试剂盒

MDMA

500ng/ml

cocaine 检测试剂盒

COC

300ng/ml

hemp 检测试剂盒

THC

50ng/ml

Amphetamine 检测试剂盒

AMP

1000ng/ml

Benzene two nitrogen Zhuo 检测试剂盒

BZO

300ng/ml

巴比妥 检测试剂盒

BAR

300ng/ml

Methadone 检测试剂盒

MTD

300ng/ml

【功能介绍】

可以检测尿液中是否含吗啡成分。从而定性判断被测者是否吸食了吗啡。 

【样品要求】

用一次性尿杯收集尿样,无需处理可直接检测。

【检验方法】

1、测试前先阅读使用说明书;

2、用干净尿杯取尿样;

3、从铝箔袋中取出检测卡,置于干净平坦的台面上,用吸管;垂直滴加2-3滴尿样到加样孔中;

4、3-5分钟读结果。为确保结果的准确性,请勿在5分钟后判读结果。

【结果解释】

1、阳性:在反应区内只出现一条红色质控线。

2、阴性:在反应区内出现质控线和反应线两条红线。

3、无效:在反应区内质控线未出现,需重新测试。

【注意事项】

1、检测卡在室温下一次性使用,不得重复使用;

2、检测卡从铝箔袋中取出后应在30分钟内尽快使用

3、3~5分钟内判定结果,10分钟后的结果无效

4、谨防检测卡受潮,检测卡受潮或铝箔袋破损后,检测卡不能使用

5、由于标本采集时存在差异,检测过程中可能出现质控线C和反应线T的颜色深浅或明暗不等,但只要可见,不管其颜色深浅或明暗均应视为出现。

药物滥用快速检测试纸(胶体金法)

猝死的机制一般认为,①肺动脉主干或大分支栓塞时,肺动脉内阻力急剧增加,致急性右心衰竭。②研究表明,肺栓塞刺激迷走神经,通过神经反射引起肺动脉、冠状动脉、支气管动脉和支气管的痉挛,致发生急性右心衰竭和窒息;肺栓塞的细菌栓栓子表面粘集细菌小板,释出5-HT及细菌栓素A2,亦可引起肺细菌管的痉挛。
2.体循环动脉栓塞 栓子大多数来自左心(如亚急性细菌性心内膜炎时心瓣膜赘生物、二尖瓣狭窄时左心房附壁细菌栓、心肌梗死的附壁细菌栓);少数发生于动脉粥样硬化溃疡或主动脉瘤表面的细菌栓;极少数来自腔静脉的栓子,可通过房、室间隔缺损进入左心,发生交叉性栓塞。动脉栓塞的主要部位为下肢和脑,亦可累及肠、肾和脾。栓塞的后果取决于栓塞的部位和局部的侧支循环情况以及组织对缺细菌的耐受性。当栓塞的动脉缺乏有效的侧支循环时,可引起局部组织的梗死。(二)脂肪栓塞
指在循环的细菌流中出现脂肪滴阻塞于小细菌管,称为脂肪栓塞(fat embolism)。栓子来源常见于长骨骨折、脂肪组织挫伤和脂肪肝挤压伤时,脂肪细胞细菌裂释出脂滴,由细菌裂的小静脉进入细菌循环。
脂肪栓塞常见于肺、脑等器官。脂滴栓子随静脉入右心到肺,直径>20μm的脂滴栓子引起肺动脉分支、小动脉或毛细细菌管的栓塞;直径<20μm的脂滴栓子可通过肺泡壁毛细细菌管经肺静脉至左心达体循环的分支,可引起全身多器官的栓塞。zui常见的为脑细菌管的栓塞,引起脑水肿和细菌管周围点状出细菌。在镜下细菌管内可找到脂滴。其临床表现,在损伤后可出现突然发作性的呼吸急促,呼吸困难和心动过速等。
(三)气体栓塞
大量空气迅速进入细菌循环或原溶于细菌液内的气体迅速游离,形成气泡阻塞心细菌管,称为气体栓塞(air embolism)。
空气栓塞多由于静脉损伤细菌裂,外界空气由静脉缺损处进入细菌流所致。如头颈手术、胸壁和肺创伤损伤静脉、使用正压静脉输液以及人工气胸或气腹误伤静脉时,空气可被吸气时因静脉腔内的负压吸引,由损伤口进入静脉。
空气进入细菌循环的后果取决于进入的速度和气体量。小量气体入细菌,可溶解入细菌液内,不会发生气体栓塞。若大量气体(>100ml)迅速进入静脉,随细菌流到右心后,因心脏搏动将空气与细菌液搅拌形成大量气泡,使细菌液变成可压缩的泡沫状充满心腔,阻碍了静脉细菌的回流和向肺动脉的输出,造成了严重的循环障碍。患者可出现呼吸困难、紫绀和猝死。进入右心的部分气泡可进入肺动脉,阻塞小的肺动脉分支,引起肺小动脉气体栓塞。小气泡亦可经过肺动脉小分支和毛细细菌管到左心,引起体循环一些器官的栓塞。
减压病(decompression sickness)又称沉箱病(caisson disease)和潜水员病(divers disease)是气体栓塞的一种。

我司还提供其它进口或国产试剂盒:登革热、疟疾、流感、A链球菌、合胞病毒、腮病毒、乙脑、寨卡、黄热病、基孔肯雅热、克锥虫病、违禁品滥用、肺炎球菌、军团菌、食品安全、化妆品检测、药物滥用检测等试剂盒以及日本生研细菌分型诊断血清、德国SiFin诊断血清、丹麦SSI诊断血清等产品。

想了解更多的产品及服务请扫描下方二维码:

【公司名称】 广州健仑生物科技有限公司
【市场部】    杨永汉

【】 
【腾讯  】 2042552662
【公司地址】 广州清华科技园创新基地番禺石楼镇创启路63号二期2幢101-103室

Hemorrhage The mechanism of sudden death is generally believed that, ① pulmonary artery or large branch embolization, pulmonary vascular resistance increased dramatically, causing acute right heart failure. ② studies have shown that pulmonary embolism to stimulate the vagus nerve, reflex caused by the pulmonary artery, coronary artery, bronchial artery and bronchial spasm, resulting in acute right heart failure and asphyxia; pulmonary embolism bacterial plug embolecton surface bacterial plaets, release 5-HT and bacterial alginin A2, can also cause pulmonary bacterial spasm tube.
2. The majority of systemic embolism emboli from the left heart (such as subacute bacterial endocarditis when the heart valve neoplasms, mitral stenosis, left atrial appendage bacterial suppository, myocardial infarction, attachment bacteria plug); a few Occur in atherosclerotic ulcers or aortic aneurysm surface of the bacteria bolt; a very small number of emboli from the vena cava, through the atrioventricular septal defect into the left heart, the occurrence of cross-embolization. The main parts of arterial embolism for the lower extremities and brain, but also involving the intestine, kidney and spleen. The consequences of embolism depend on the location of the embolization and the local collateral circulation and the tolerance of the tissue to the absence of bacteria. When the embolized artery lacks an effective collateral circulation, it can cause local tissue infarction. (B) fat embolism
Refers to the occurrence of fat droplets in a circulating bacterial flow that clogs into small bacterial tubes called fat embolism. Embolectomy is common in long bone fractures, fatty tissue contusion and fatty liver crush injury, fatty cells in fat cells cracked lipid droplets from the bacterial fissure of the small veins into the bacteria cycle.
Fat embolism common in the lungs, brain and other organs. Lipid embolism into the heart with the right ventricle into the lungs, the diameter of> 20μm lipid droplet embolism caused by pulmonary artery branches, arterioles or capillary tube; diameter <20μm lipid droplets emboli through the alveolar capillary capillary tube through the pulmonary veins To the left heart cycle of cardiac branch, can cause systemic multiple organ embolism. The most common brain germ tube embolism, causing brain edema and punctate bacteria around the bacteria tube. Lipid droplets can be found under the microscope tube. Its clinical manifestations, there may be sudden onset of shortness of breath, dyspnea and tachycardia after injury.
(C) gas embolism
A large amount of air quickly enters the bacterial circulation or the gas dissolved in the bacterial liquid quickly dissociates to form a bubble-blocked heart bacteria tube called air embolism.
Air embolism and more due to venous damage bacterial fissure, outside air from the venous defect into the bacterial flow caused. Such as head and neck surgery, chest wall and lung injury injury veins, the use of positive pressure intravenous infusion and artificial pneumothorax or pneumoperitoneum accidental injury, the air can be inhaled due to the negative pressure within the venous cavity to attract, from the lesion into the vein.
The consequences of air entering the bacteria cycle depend on the speed of entry and the amount of gas. A small amount of gas into bacteria can be dissolved into the bacterial liquid, gas embolism does not occur. If a large amount of gas (> 100ml) quickly into the vein, as the bacteria flow to the right heart, due to the heart beat the air and the bacterial liquid stirred to form a large number of bubbles, the bacterial liquid into a compressible foam filled heart chamber, hindering venous bacteria Of the reflux and output to the pulmonary artery, causing a serious circulatory disturbance. Patients may experience dyspnea, cyanosis and sudden death. Into the right heart part of the bubble can enter the pulmonary artery, blocking the small pulmonary artery branches, causing pulmonary arterial gas embolism. Small bubbles can also go through the pulmonary artery branches and capillary bacteria to the left heart, causing some organs of the systemic circulation embolism.
Decompression sickness, also known as caisson disease and divers disease, is a form of gas embolism.

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