美国Alfa 瘦肉精多联快速检测卡

广州健仑生物科技有限公司

我司长期供应瘦肉精三联检测卡，本产品用于快速检测动物尿样、组织和饲料中盐酸克伦特罗、莱克多巴胺、沙丁胺醇残留，整个检测过程只需要3-5分钟左右，具有操作简单，方便快捷，灵敏度高特异性强等特点。

瘦肉精检测试剂进口品牌：美国Alfa、美国US

我司还提供其它进口或国产试剂盒：登革热、疟疾、流感、A链球菌、合胞病毒、腮病毒、乙脑、寨卡、黄热病、基孔肯雅热、克锥虫病、违禁品滥用、肺炎球菌、军团菌、化妆品检测、食品安全检测等试剂盒以及日本生研细菌分型诊断血清、德国SiFin诊断血清、丹麦SSI诊断血清等产品。

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【瘦肉精的危害】

“瘦肉精”进入动物体内后主要分布于肝脏。肌肉中含量较肝脏低很多。人摄入后在体内存留时间较长，其不良反应主要有：可引起心率加速，特别是原有心律失常的病例更易发生心脏反应，可见心室早搏、ST段与T波幅压低，还会发出肌肉震颤，引发四肢、面颈部骨骼肌震颤，尤其是交感神经功能亢进的病例更易发生。此外，还可引起代谢紊乱、血钾降低，引起心慌、肌肉震颤、头痛以及脸部潮红等。对心率失常、高血压、青光眼、糖尿病、甲状腺机能亢进等疾病的患者有较大危害。

美国Alfa 瘦肉精多联快速检测卡

【产品简介】

本产品为克伦特罗-莱克多巴胺-沙丁胺醇三合一胶体金快速检测卡，用于定性检测猪、牛、羊尿液、组织和饲料中的瘦肉精残留，整个检测过程只需要3-5分钟左右。

【检测限】

克伦特罗3ng/ml（3ppb），莱克多巴胺3ng/ml（3ppb），沙丁胺醇3ng/ml（3ppb）

【产品组成】

克伦特罗-莱克多巴胺-沙丁胺醇三合一胶体金快速检测卡（40T/盒）

滴管（1个/袋）、干燥剂（1片/袋）

【样品处理】

用干燥、洁净的离心管或适当容器采集50ml左右尿液。如果不立即检测，待检样本在2-8℃存放，可保存24小时，注意避免腐坏造成失效或污染。出现阳性结果应按法定程序分瓶封装样品用于确证法检测。

【使用步骤】

1、测试前先完整阅读说明书，使用前将检测卡和待检样本溶液恢复至室温4～30℃。

2、从原包装袋中取出检测卡，打开后请在一个小时内尽快地使用。

3、将检测卡平放，用滴管吸取待检样品溶液，缓慢垂直滴加2-3滴于加样孔中，加样后开始计时。

4、结果应在3-5分钟时读取，其他时间判读无效，根据示意图判定结果。

【结果判断】

1. 阴性（-）：两条紫红色条带出现。表示样品中不含有瘦肉精或其浓度低于检测限。
2. 阳性（+）：检测T线无显色，则表示样品中瘦肉精浓度高于检测限。

1. 无效：未出现质控C线，表明操作过程不正确或检测卡已失效。

【注意事项】

1、检测卡请在保质期内一次性使用；

2、检测时避免阳光直射和电风扇直吹；

3、尽量不要触摸检测卡中央的白色膜面；

4、采样滴管不可混用，以免交叉污染；

5、如果待检样本出现沉淀或浑浊物，请离心后再检测；

6、试验遇到的任何问题，请与供应商。

【储存及有效期】

原包装应储存于4～40℃，阴凉避光干燥处，切勿冷冻；有效期24个月。有效期及批号见外包装。

美国Alfa

C型产气荚膜梭菌不仅感染猪，还可感染羊、鸡、马、兔和牛等动物。健康动物肠道内存在该菌，一旦动物肠道内的菌群失调，产气荚膜梭菌大量繁殖和生长，排出的粪便污染了动物饲料或违禁品。大量的该病菌就会随着幼畜吸吮奶或食用污染物等方式进入幼畜体内，侵袭肠绒毛上皮并产生大量毒素，使肠道正常组织破损，毒素趁机而入，进而随血液循环侵害其它组织器官。
实验室中由C型产气荚膜梭菌所导致的动物和人类的肠毒血症和肠炎，与自然感染的动物临床症状相似，病程也可分为亚急性、慢性和急性。其中急性和亚急性病例常表现为精神抑郁、剧烈腹痛和带血稀便。慢性病例主要发生在成年动物中，其临床表现为脱水和持续性腹泻。
D型产气荚膜梭菌能产生α、ε毒素，可导致成年牛的肠毒血症、山羊的小肠和结肠炎、犊牛和羔羊的肠毒血症（软违禁品）等。肠毒血症是由D型产气荚膜梭菌而引起的一种重要的人畜共患病。该病的流行特点是发病急骤，死亡快，多呈散发，可以引起发病动物的大量死亡，严重制约了畜牧业的长远发展。
感染D型产气荚膜梭菌的绵羊和山羊两者临床表现不同，山羊主要表现为小肠结肠炎，而绵羊病理变化表现为心包积水、大脑和肺部水肿、肠道症状。以上两者的实质器官病理变化相似。
D型产气荚膜梭菌在动物肠道内不断增殖和产生大量ε毒素，高浓度毒素破坏了肠道壁的完整性，使肠道的通透性大大提高，毒素大量进入血液循环系统，甚至损害与生命活动相关的神经元（如脑干），进而导致患病动物休克或死亡。
肠毒血症的潜伏期较短，少见临床表现。临床上依据动物肠道吸收毒素多少常将该病分为两种类型：一种是以强烈抽搐为特征，一般在 2-4 h内死亡，患该病动物死亡前肌肉抽搐，流涎过多，四肢不停滑动，眼珠翻转，磨牙，随后头颈明显抽缩；另一种是以昏迷和嗜睡为主要特征，病程缓慢，早期临床症状表现为流涎，上下颌“咯咯”作响，之后昏迷，角膜反射等。该病的病理变化常限于呼吸系统、消化系统及心血管系统。剖检常发现患该病的动物真胃含有未消化的食物，回肠部分呈现急性出血性炎症变化，重症病例整个肠段因严重充血而呈红色。
由D型产气荚膜梭菌所引发的动物肠毒血症有明显的条件性和季节性，秋季牧草结籽后或春末夏初青草萌发是牧区动物产气荚膜梭菌病的多发期。在农业地区，该病常发生于庄稼收获季节或收菜季节，原因是在该季节动物常食入过量的谷物、菜叶等，造成肠道消化不良，从而进一步导致肠道菌群失调，为肠道内有害菌群的大量繁殖提共了可乘之机。

美国Alfa

我司还提供其它进口或国产试剂盒：登革热、疟疾、流感、A链球菌、合胞病毒、腮病毒、乙脑、寨卡、黄热病、基孔肯雅热、克锥虫病、违禁品滥用、肺炎球菌、军团菌、食品安全、化妆品检测、药物滥用检测等试剂盒以及日本生研细菌分型诊断血清、德国SiFin诊断血清、丹麦SSI诊断血清等产品。

想了解更多的产品及服务请扫描下方二维码：

【公司名称】 广州健仑生物科技有限公司
【市场部】    杨永汉

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【腾讯  】 2042552662
【公司地址】 广州清华科技园创新基地番禺石楼镇创启路63号二期2幢101-103室

Clostridium perfringens type C not only infect pigs, but also in sheep, chickens, horses, rabbits and cattle and other animals. The bacteria exist in the intestine of healthy animals. Clostridium perfringens multiplies and grows once the flora in the intestine of the animal is out of control. The excreted faeces contaminate animal feed or contraband. A large number of the bacteria will be with young animals suck milk or food contaminants into the young animals such as the body, invasion of intestinal villi and produce large amounts of toxins, the normal tissue damage to the intestine, toxin took the opportunity to enter, and then infringement of blood circulation with other Tissue organs.
Animal and human enterotoxemia and enteritis caused by Clostridium perfringens type C in laboratories are similar to the clinical symptoms of naturally infected animals and their duration can be sub-acute, chronic and acute. The acute and subacute cases often show depression, severe abdominal pain and loose stools. Chronic cases occur mainly in adult animals, with clinical manifestations of dehydration and persistent diarrhea.
Clostridium perfringens type D can produce alpha, epsilon toxins that can cause enterotoxemia in adult cattle, small intestine and colitis in goats, enterotoxemia (soft contraband) in calves and lambs, and the like. Enterotoxemia is an important zoonosis caused by Clostridium perfringens type D. The prevalence of the disease is characterized by sudden onset, rapid death, mostly distributed, can cause a large number of deaths of animals, seriously hampered the long-term development of animal husbandry.
The clinical manifestations of both sheep and goat infected with Clostridium perfringens type D were different. Goat mainly manifested as enterocolitis. Pathological changes in sheep showed pericardial hydrops, brain and pulmonary edema, and intestinal symptoms. Both of the above two organs of the same pathological changes.
Clostridium perfringens type D proliferates and produces a large amount of epsilon toxin in the intestine of animals, and high levels of toxins disrupt the integrity of the intestinal wall, greatly increase the intestinal permeability, large amounts of toxins enter the blood circulation, and even Damage to life-related neurons (such as the brain stem), which in turn leads to the shock or death of diseased animals.
The incubation period of enterotoxemia is shorter, rare clinical manifestations. Clinically based on animal intestinal absorption of toxins how often the disease is divided into two types: one is characterized by strong convulsions, usually within 2-4 h of death, suffering from animal disease before the onset of muscle twitching, salivation too much, The other is based on coma and lethargy as the main feature of the slow course of the early clinical manifestations of salivation, the mandibular "giggle" sound, then coma, corneal reflex Wait. Pathological changes of the disease is often limited to the respiratory system, digestive system and cardiovascular system. Autopsy found that the animals suffering from the disease really contain undigested food, ileum part of the acute hemorrhagic inflammation showed changes in severe cases of intestinal bowel due to severe congestion and red.
Animal enterotoxemia caused by Clostridium perfringens type D has obvious condition and seasonality. The grazing of grass in autumn or early summer after the autumn grazing is the frequent occurrence of Clostridium perfringens disease in pastoral animals period. In agricultural areas, the disease often occurs during the harvest season or during the harvest season because animals eat excess grains, leaves, etc. during the season, causing intestinal dyspepsia, which in turn leads to an imbalance in the gut flora A large number of harmful bacteria within the road to mention a large number of opportunities available.